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Maternal immune activation impacts on the kynurenine pathway in preadolescent offspring: Preventive effects of anti-inflammatory treatment.

Katerina Zavitsanou

Kynurenic acid (KYNA), a glia-derived metabolite of the kynurenine pathway (KP) of tryptophan degradation, is an antagonist at glutamate NMDA and a7 nicotinic acetylcholine receptors. Abnormalities in the KP have been described in patients with schizophrenia. Cyclo-oxygenese-2 (COX-2) inhibitors show considerable promise in treating early (but not late) stage schizophrenia and preclinical studies suggest that COX-2 inhibitors decrease KYNA in the brain. 

12th Biennial Australasian Schizophrenia Conference
13-14 May 2013 Melbourne, Australia.
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VideoMaternal immune activation impacts on the kynurenine pathway in preadolescent offspring: Preventive effects of anti-inflammatory treatment.Katerina Zavitsanou12'38"
icon pdf.gifMaternal immune activation impacts on the kynurenine pathway in preadolescent offspring: Preventive effects of anti-inflammatory treatment.Katerina Zavitsanou 


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Katerina Zavitsanou

Dr Katerina Zavitsanou, School of Psychiatry, University of New South Wales, Neuroscience Research Australia, Schizophrenia Research Institute. Katerina spent 4 years (2000-2004) in the School of Biomedical Science, University of Wollongong, where she contributed in the establishment of a novel research program in schizophrenia research funded by the Schizophrenia Research Institute. Her research focused on the investigation of neurochemical abnormalities in post mortem brain of schizophrenia patients and in animal models of disease. She accepted a position with ANSTO in 2004 to establish a laboratory for in vitro histological, light microscopy and autoradiographic techniques to be used in the evaluation of new radiopharmaceuticals.

Co-authors: Dr Chai K. Lim, Dr Tertia Purves-Tyson, Ms Katherine M. Allen, Professor Gilles J. Guillemin, Professor Cynthia Shannon Weickert,
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